公司資料

Allarity Therapeutics 股價、新聞與催化劑

股票代碼:
ALLR
交易所:
NASDAQ
產業:
Biopharma
所在地:
Denmark

股票表現

1.34 USD (-2.19%)

收盤價

最後更新: 2026年8月17日

26.9M USD

市值

股價為何變動

The latest company update was “Allarity Therapeutics Reports Second Quarter 2026 Results and Completion of the Phase 3-Ready Stenoparib Manufacturing Campaign”. ALLR changed 0.74% and closed at $1.37 on Aug 14, 2026, indicating a relatively limited closing-price reaction to this clinical readout update.

公司概覽

Biomarker-Guided Oncology

Allarity Therapeutics develops personalized cancer therapies paired with drug-specific companion diagnostics. Its strategy is to identify patients whose tumor gene-expression profiles indicate a higher probability of responding to a particular anticancer medicine.

Stenoparib

Stenoparib is an oral small molecule that inhibits PARP1/2 and tankyrase1/2. This dual mechanism combines DNA-damage interference with inhibition of WNT/beta-catenin signaling, a pathway associated with tumor progression, metastasis, and drug resistance. Allarity holds exclusive global development and commercialization rights to the candidate.

DRP Companion Diagnostic

The Drug Response Predictor platform analyzes messenger-RNA expression patterns from tumor biopsies to generate a drug-specific response score. Allarity is developing a stenoparib-specific DRP companion diagnostic to enrich clinical studies for patients most likely to benefit and operates a CLIA-certified laboratory capable of performing the testing for US trials.

Ovarian-Cancer Development

Stenoparib is being evaluated in a Phase 2 study for advanced recurrent ovarian cancer that is platinum-resistant or platinum-ineligible. The program has FDA Fast Track designation, and the completed late-stage manufacturing campaign provides drug supply suitable for a future registrational trial.

Additional Tumor Programs

A Department of Veterans Affairs-funded Phase 2 study is evaluating stenoparib with temozolomide in relapsed small-cell lung cancer. Preclinical findings also support potential development in colorectal cancer through inhibition of WNT pathway signaling.

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